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Seed Oils Ruin Thyroid Function: 🟥 Reducing the basal metabolic rate 🟥 Reducing thyroid hormone binding to transport proteins 🟥 Reducing conversion to the active form of thyroid hormone 🟥 Reducing nuclear receptor binding
○ The thyroid gland produces hormones T3, the active hormone, and T4. ○ The purpose of thyroid hormone (T3) is to reach the cell's nucleus and activate the nuclear receptor. ○ When T3 activation of the nuclear receptor is high, your body is more efficient at converting the food you eat into usable energy - rather than storing it as fat. ○ 'Calories out' increases.
○ At rest, your body needs energy to maintain vital functions like breathing, circulating blood, and repairing cells. ○ The amount of calories used for these purposes is the basal metabolic rate. ○ Anything that interferes with T3 can lead to issues including fatigue, weight gain, depression, hormonal imbalances, sexual dysfunction, mental health problems, and serious cardiovascular conditions.
○ A higher basal metabolic rate (BMR) means a higher resting metabolism. ○ When rats are fed a diet very ⬇ low in seed oil fatty acids, their 🟩 BMR is significantly increased. ○ Rats fed corn oil gain more weight than those fed coconut oil. ○ Coconut oil is low in PUFAs, the class of fatty acids which most interfere with thyroid function. ○ A high seed oil diet produces "increased feed efficiency," the ability to gain more weight from the same or fewer calories.
○ In one of the few human experiments designed to deplete 'essential' fatty acids: 🟩 The metabolic rate significantly increased, and fatigue disappeared. ○ Those nominally 'essential' fatty acids are abundant in seed oils. 🟥 Regardless of their essentiality status, we are consuming way more than necessary.
○ As seed oil consumption ⬆ increases in the population, the basal metabolic ⬇ rate drops. ○ It is a common belief that we are eating more and moving less; however, in reality, our metabolisms are slowing down. ○ The drop in metabolic rate caused by consuming seed oils indicates that they inhibit thyroid hormone in some way. ○ Let's look at some of those mechanisms.
○ Thyroid hormones are transported around the body by binding proteins in blood serum. ○ A number of fatty acids reduce thyroid hormone binding in human blood serum. ○ Arachidonic acid and linoleic acid have the most potent anti-thyroid effect. ○ Linoleic and arachidonic acids prevent binding but saturated fatty acids do not. ○ Linoleic acid is the major fatty acid in seed oils like soybean oil. We consume multiple times more linoleic acid than our grandparents did. ○ Arachidonic acid comes from animal foods, and we can make it from linoleic acid. These two are the major omega-6 fatty acids.
○ Extrathyroidal conversion is the conversion of T4 to T3 outside the thyroid gland. ○ It's a critical process, as most of your T3 is not released by the thyroid gland, but must be converted from T4 elsewhere in the body. ○ Researchers discovered that the same fatty acids that interfere with thyroid hormone transport also reduce the liver's ability to convert T4 to T3. ○ Linoleic and arachidonic acids decrease thyroid hormone conversion in the liver, reducing the amount of T3 available. ○ PLA2 is an enzyme that releases fatty acids like linoleic and arachidonic acids into the blood. Increasing it results in lower thyroid hormone binding.
○ For T3 to exert most of its benefits, it must finally bind to the nuclear receptor. ○ Cell researchers investigated the effect of different fatty acids on T3 nuclear binding. ○ Linoleic, arachidonic, linolenic, and oleic acids inhibited T3 nuclear binding. ○ Linoleic and linolenic acids are two fatty acids found in seed oils. ○ Palmitoleic acid, a monounsaturated fatty acid, caused the greatest nuclear receptor binding inhibition. ○ Linoleic acid was a very close second place. ○ Saturated fatty acids exhibited little to no inhibition of nuclear receptor binding.
